By J.R. Baker (ed.), R. Muller (ed.)
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1970). The testing of proven Trypanosoma brucei and T . rhodesiense strains by the blood incubation infectivity test. Bulletin of the World Health Organization 42, 91 1-9 16. Schiitt, I. D. and Mehlitz, D. M. (1982). On the persistence of human serum resistance and isoenzyme patterns in experimentally infected pigs. Acta Tropica 38, 367-373. Scott, C. M. (1981). Mixed populations of Trypanosoma brucei in a naturally infected pig. Tropenmedizin und Parasitologie 32, 22 1-222. Scott, C. -L. Toudic, A.
1980) are included in this strain group; their dendrogram revealed a marked degree of separation between these zymodemes and those now placed in kiboko (see Section IV B 3). More recently, Gibson et a f . (1985) placed a ZI 1 isolate (J lo), on the evidence of its kDNA maxi-circle polymorphisms, into kiboko. This isolate, however, differed in possessing an extra, unique, restriction site, which may support its removal from kiboko into kakumbi. Since the mammalian hosts in kakumbi were all wild animals, it may be that it is an East African form of T.
BAKER, L. R. RICKMAN A N D D . MEHLITZ patients in and around the Luangwa Valley in Zambia. A strong link with human infectivity existed, since the zymodemes contained 97% (185/191) of the populations originating from patients in Zambia; similar populations were isolated from animals in the same collecting area. , 33 in 2 2 , 2 2 in 2204, and I7 in 23). Twenty of the unrecognized zymodemes from Zambia were in the sindo (Section IV B 2) or kakumbi (Section IV B 4) strain groups. Most zymodemes were in sets 6 , 9 and 10.